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酒精对健康的影响 Alcohol

 苍穹月光 2022-11-16 发布于湖南

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一直以来,我们对喝醉酒的印象是口齿不清、无法直线行走、无法清晰思考或说话。如果这一切变得更严重,一个人会呕吐或丧失能力,我们称之为酒精中毒。但我们真的知道当血液充满大量酒精时会发生什么吗?我们不知道,我们也从来没有知道过真相。
科学研究假设说,血液中大量的酒精会直接导致上述行为和症状,即酒精本身会影响大脑,导致醉酒行为。实际上没有人准确知道酒精对大脑的影响,而且这些醉酒表现只是一个人喝酒时对大脑影响的一个方面。
酒精的影响始于微醉。有人说酒精可以帮助他们放松,并说出自己的想法。有人说几杯啤酒帮助他们放松。有人说葡萄酒有助于他们处理情绪。然而,酒精是如何让他们“放松”或“处理情绪”的?

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一.大脑骗局游戏
我们的大脑依靠葡萄糖生存。大脑缺乏葡萄糖会导致饥饿。如果我们去除一个人血液中葡萄糖,葡萄糖未能进入大脑,一个人可能会在短时间内陷入大脑衰竭。酒精是大脑的终极骗局。
因为大脑会认为酒精是糖,认为是大脑可以使用的糖。它认为酒精和包含碳水和糖分的食物中的葡萄糖一样。实际上酒精是甲基糖,更准确地说,这是一种汽化糖,而不是一种可用的糖。酒精的本质是糖,但它却不是糖。
这场骗局会在大脑上演,导致大脑将酒精认为是急需的葡萄糖,从而导致问题。血液中的酒精越多,大脑就越难利用血液中任何真正的葡萄糖来源。酒精会让大脑中任何剩余的葡萄糖储存都站到一边去,酒精成为主导地位。
我们的肝脏是人体主要的葡萄糖储备所在,其工作是在大脑需要时正确地释放葡萄糖。有时我们的饮食中缺乏糖、葡萄糖、果糖或任何种类的碳水化合物,血液中出现葡萄糖不足。在这种情况下,肝脏的工作是释放葡萄糖,这样大脑就不会完全饿死。在一个人长时间不进食的情况也是如此,要知道,如果没有葡萄糖,大脑将无法生存。正是因为肝脏的这个重要功能,所以一个人在做清水断食时能活下来,因为肝脏在为大脑释放充足的葡萄糖。
大脑中的糖原储备和肝脏中的葡萄糖储备决定了一个人可以断食多长时间。现在几乎每个人都存在肝脏停滞,许多人的葡萄糖储备很少,因此饮酒对这些人的影响比对肝脏强健的人影响更大。这就是为什么有人说“我喝不了酒”的原因。尤其是随着年龄的增长,一个人肝脏变得越来越弱。
不管一个人对酒精的耐受性如何,当摄入大量的酒精时,对人的影响都是一样的。当一个人喝酒时,这会导致肝脏中毒和变得麻木,肝脏是阻止酒精进入大脑的防御机制。当一个人开始微醉时,或我们可能将这称为感觉到酒精作用的开始阶段,无论这种反应是多么温和,我们的肝脏已经处于保护大脑的饱和水平。我们理解的所谓饮酒适度的状态,实际上肝脏早就到了饱和状态,了解这一点非常重要。
任何数量的酒精都是有毒的,因此肝脏的工作就是吸收它所能吸收的每一滴酒精。当肝脏被酒精毒害时,就无法再释放葡萄糖了。即使一个人有很充足的葡萄糖储备,最终肝脏也会因为酒精而导致这一重要功能的瘫痪,肝脏停止释放葡萄糖,相反,它的工作变成了吸收酒精。
同时,酒精会让血液中的任何葡萄糖都站到一边去,使得酒精成为大脑所需燃料的首要选择,因为酒精看起来像葡萄糖,即使实际上并不是如此。酒精是糖的副产品,就像糖曾经的鬼魂。大脑会成为酒精幽灵效应的受害者。随着越来越多的酒精进入大脑,一个人会表现出更多的醉酒行为。当一个人达到醉酒行为的地步:口齿不清、无法正常运作,这意味着大脑已经开始快饿死了。理解这一点至关重要:我们所知道的醉酒和酒精中毒相关的大多数症状不仅仅源于酒精本身,大多数症状实际上是大脑开始死亡的症状。
一个人在晚上或白天喝的酒越多,大脑能够吸收为其所用的葡萄糖就越少,假设最佳状态是100%的葡萄糖进入大脑并保持其活力,那么饮酒会使葡萄糖的百分比下降到5%到10%,取决于一个人醉酒的程度。这就像把一条鱼从水里捞出来,看着它在沙滩上喘息,因为它吸的是氧气而不是水,然后把它放回水中使其复活,然后再把它捞出来,如此重复。鱼会活下来,但它会因为这种生存模式而付出代价。一个人经常饮酒时会发生这种情况。有一点点葡萄糖进入大脑以维持人的生命,但量少到一个人失去了运作能力,变成了一个会走路说话的濒死大脑的例子。
不仅肝脏会因为酒精中毒而瘫痪,无法释放足够的葡萄糖进入大脑,而且酒精会屏蔽其他进入大脑的葡萄糖,大脑会选择酒精而不是葡萄糖。这并不是因为大脑需要酒精,也不是因为酒精对大脑有好处。只是因为大脑被欺骗认为酒精是最容易获得的可用葡萄糖形式。

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二.酒精中毒
酒精对大脑的影响不仅仅是上述葡萄糖骗局的结果,即大脑变得缺乏真正有用的葡萄糖。酒精的确是一种毒药,作为毒药,它确实具有令人陶醉和让人衰弱的作用。然而,当一个人喝第三杯酒时,口齿不清是因为大脑开始因缺乏葡萄糖而挨饿,从而失去了运作的能力。
而当一个人的酩酊大醉变得极端和猛烈时,喝到昏倒并睡着了,这种醉酒的睡眠实际上是俄罗斯转盘游戏(注:俄罗斯转盘游戏是在手枪的六个弹槽中放入一颗或多颗子弹,任意旋转之后,把手枪对着自己的头,扣动扳机,意味着这是一场性命的赌博)。因为如果由于严重酒精中毒,大脑没有得到任何葡萄糖,大脑会饿死,一个人可能会在睡梦中死去。或者由于酒精中毒,他们可能想要呕吐(这不是大脑缺乏葡萄糖引起的症状)。由于大脑会因缺乏葡萄糖而死亡,神经无法运作。当大脑失去更深层的葡萄糖储备时,迷走神经可能会瘫痪,这意味着一个人可能会在睡眠中呕吐,这会更容易窒息和死亡。
在饮酒之前摄入足够的富含葡萄糖食物并保持低脂饮食很重要,这样一个人可以有足够的新鲜葡萄糖储备。这就是为什么如果一个人空腹喝酒,没吃东西,他会更容易更快醉酒,更快出现醉意。我们以为醉酒是酒精导致的,实际上这是大脑缺乏葡萄糖的结果。肝脏开始吸收酒精,无法再释放葡萄糖,而且由于一个人饮酒前没有吃东西,血液中缺乏新鲜的葡萄糖。对于饮酒前吃过东西的人来说,当他们开始饮酒时,酒精需要更长的时间才能影响他们,因为他们至少还有大脑所需的新鲜葡萄糖。
酒精中毒需要更大量的酒精才能发生,也就是说,真正是酒精中毒导致的症状出现需要更大量的酒精。即便如此,醉酒部分是酒精中毒导致的,部分是大脑缺乏葡萄糖导致的。
如果是酒精中毒导致的醉酒,症状是有限的:一个人会恶心、呕吐、头晕,但尽管很不舒服,但他们仍然是清醒的。然而正是因为大脑缺乏葡萄糖在饿死时,才会导致一个人功能丧失、口齿不清和说话困难,或不理解别人在说什么,自己也不知道自己在说什么。由于大脑获得的葡萄糖非常少,在维持生命的边缘,大脑的某些部分开始关闭。

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三.错上加错的宿醉疗法
宿醉,就像醉酒本身一样,部分是大脑匮乏葡萄糖在饿死导致的,部分是酒精中毒导致的。最糟糕的恢复方法是第二天继续喝酒。尽管这是给许多喝酒的人的建议,但这真的是最糟糕的选择。这无法起到作用,无法摆脱宿醉,因为大脑再次会因为匮乏葡萄糖而挨饿。
许多人酒后第二天大吃大喝的原因是他们的大脑亟需葡萄糖。取决于一个人喝了多少酒,大脑因缺乏葡萄糖而差点死亡,所以现在大脑会向全身发出信息,它立即迫切需要大量的葡萄糖。同时,有些人仍然可能因酒精而感到恶心、胃部不适,好像吃不下食物,宿醉中这部分反应是酒精的毒性导致的。
许多人不会喝到恶心和呕吐的程度。他们会想要很快吃下大量食物以醒酒。他们会在喝酒的同一天晚上,在凌晨两点或五点,点一大堆吃的。或者他们第二天会持续吃很多东西,号称是为了吸走酒精。甚至当有人在聚会或酒吧里大量饮酒时,同伴会说“你必须吃点东西来吸走酒精”。
没有人意识到,酒后想要进食实际上是大脑需要葡萄糖,使其摆脱饥饿状态并重新开始运作。但因为我们不了解身体所需,我们依然没有给予大脑真正需要的东西,因为我们通常会选择摄入糖分(碳水化合物)和脂肪的组合。而油脂或脂肪会抑制碳水化合物中的葡萄糖进入大脑。例如,薯条中的油脂会阻碍土豆中的糖分进入大脑。这会导致胰岛素抗性,身体必须付出极大的努力将糖分送入大脑和身体其他急需的部位。我们真正需要的是富含葡萄糖的水果诸如香蕉,或生菠菜或其他绿叶蔬菜,或西芹汁等富含矿物盐的食物以从醉酒的神志不清中恢复过来。相反,我们通常会选择油脂和糖分的组合,诸如披萨、薯条、米饭加炒菜、面条加油炒的浇头等,这些都是碳水化合物(糖分)和油脂的组合,油脂会阻碍糖分顺利进入大脑。

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四.肾上腺素激增
为什么酒精会让人上瘾?因为当大脑失去了维持生命的燃料葡萄糖时,会出现意想不到的肾上腺素飙升。进入大脑的酒精越多,大脑越匮乏葡萄糖,肾上腺素分泌就越多。因为身体会分泌肾上腺素以代替缺失的糖分,作为应急。
当我们说酒精让人说话时,我们真正目睹的是肾上腺素在被饿死的大脑使用。每当我们处于任何程度的危险中时,我们的肾上腺都会释放出一种肾上腺素混合物,以改变我们血液中的化学成分,以提供任何可能的帮助。当大脑没有燃料时,肾上腺素会成为备用燃料。请记住,肾上腺素本身会让人上瘾。我们酒喝得越多,大脑就越缺乏葡萄糖燃料,身体释放的肾上腺素就越多。这也是造成一个人酒精上瘾的原因之一。
这种肾上腺素对每个人的影响不同。这决定了一个人会是愤怒的醉酒者,还是在喝醉时坐在地上哭泣,或大喊大叫。这种肾上腺素激增会因为人们不同生活经历而产生不同的影响。当肾上腺素被释放时,我们每个人都会感受到不同的情绪。当一个人喝酒时,过去的生活经历和伤口往往会浮现。这就是为什么每个人在喝酒时会有不同情感体验的原因之一。有人说酒精让他们放松,有人说酒精导致偏头痛,有人说酒精让他们感觉悲伤和沮丧,有人说酒精使其快乐,有人说酒精给他们力量、能量和勇气。这完全取决于一个人对肾上腺素激增的反应。有些人在开始喝酒时会感到兴奋、尖叫,在第一轮啤酒时就大喊大叫,无论是在体育赛事中欢呼,还是只是庆祝那个时刻。
所谓的“欢乐时光”(happy hour)是有原因的。当一个人喝第一杯酒时产生的感觉实际上是面对大脑缺乏葡萄糖饿死时肾上腺素激增的感受。如果我们理解这一点,我们不会喊“干杯!”,而会说:“我的大脑快要饿死了!我的肾上腺会释放出大量的肾上腺素来维持我的大脑活力!与此同时,我将感受到酒精这样毒药对大脑的影响!这将给我一个美好的夜晚!”,实际上酒精永远无法让你拥有一个美好的夜晚。

有酒瘾的人恰恰是因为其大脑非常迫切需要葡萄糖。酒精会迅速进入大脑,带来快感,但这会使人付出巨大的健康代价。与水果和淀粉类蔬菜中的葡萄糖不同,酒精中的甲基糖会随着时间“腌制”大脑,就类似于用醋腌制黄瓜一样的效果。酒瘾其实反映的是大脑迫切需要葡萄糖,在饮食中加入大量水果是摆脱酒瘾至关重要的一步。

同时,许多有酒瘾的人并不一直想要进食。许多有酒瘾的人可以很长时间不吃东西,或吃得很少,而是依靠酒精获取每日活动所需的卡路里,这是因为酒精上瘾导致的体内大量肾上腺素分泌会抑制胃口,让人不想进食。应对酒瘾最重要的是每隔一两个小时吃一些水果和蔬菜的组合(请避开与油脂和脂肪同时摄入),不仅可以补充身体缺乏的葡萄糖,而且可以避免身体分泌过量肾上腺素,椰枣、芒果或非转基因木瓜都是对抗酒瘾极其有帮助的食物。

更多关于上瘾的原因信息,请见
上瘾的真相(上)(酒瘾、烟瘾、糖瘾、盐瘾、食物上瘾、贪食症和厌食症、药物上瘾、极限运动瘾等)(下)
更多关于糖分和油脂为什么要分开摄入的原因,请见:
健康误区(37)- 油脂和脂肪的迷思
新手如何开始实践安东尼威廉净化法,请见:
新手指南
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Alcohol
Throughout the centuries, all the way up through today, we have associated someone who’s had too much to drink with slurred speech, the inability to walk in a straight line, the inability to think or speak clearly. We call it being drunk. If it becomes more severe and that person is vomiting or incapacitated, we may call it alcohol poisoning. Do we really know what happens when our bloodstream is saturated with a high volume of alcohol? We don’t, and we never did.
We assumed, because medical research and science assume, that more alcohol in the bloodstream leads directly to these actions and symptoms— that alcohol itself affects the brain to cause these experiences. Here’s the truth: no one knows exactly what happens inside the brain with alcohol. Intoxication is not fully understood; it’s only one aspect of what happens to the brain when someone drinks.
Alcohol’s effects start out with what is called getting a little “tipsy.” Some say alcohol helps them loosen up and speak their mind. Some say a couple of beers help them relax. Some say wine helps numb them out. Yethow does alcohol seem to loosen them up, relax them, or numb them out? 
BRAIN CHARADES
As we’ve discussed throughout this book, your brain survives on glucose. A lack of glucose to your brain can slowly starve it over time. And if glucose were eliminated from your bloodstream and never entered your brain, you could go into brain failure within moments. Alcohol is the all-time trick on your brain. That’s because your brain believes alcohol is sugar, sugar it can use. It believes it’s like the glucose that’s created by the foods you eat that contain carbohydrates and sugar. Really, alcohol ismethyl-sugar. It’s a hybrid of whatwas sugar—more of a vaporized sugar versus a usable sugar. Alcohol’s essence is sugar, yet it’s not. 
As a result of this charade played on your brain that leads it to identify alcohol as critically needed glucose, several things go wrong. The more alcohol in your bloodstream, the harder it is for your brain to use any real source of glucose in your bloodstream. And alcohol dominates over any glucose storage left in the brain.
Your liver is your main glucose storage bin. Its job is to release glucose congruently as your brain needs it. There are moments when we aren’t getting sugar or glucose or fructose or any kind of carbohydrate in our diet, and we go into glucose deficits in the bloodstream. Your liver’s job in that case is to release glucose so your brain doesn’t completely starve. This includes circumstances where you aren’t eating for a long period of time. Remember, without glucose, your brain can’t survive. The liver fulfilling this role is why someone can do a water fast and their brain can survive the experience—because the liver is releasing ample glucose for the brain. Glycogen reserves in the brain and glucose reserves in the liver determine how long someone can withstand a water fast. Now, not everybody’s liver functions that well. Many people’s glucose storage bins can be minimal due to the liver being sluggish or stagnant, so drinking alcohol can affect these people more than alcohol affects people with strong livers. That’s why some say, “I can’t handle alcohol,” or “He can’t handle his liquor.” Because their livers are weakened, especially as they age.
Regardless of someone’s alcohol tolerance, when enough alcohol is consumed, the effects are all the same. As you’re drinking alcohol, it starts to poison and numb the liver, and your liver is your defense mechanism to stop alcohol from getting to your brain. By the time you start getting tipsy—or whatever you personally call the beginning stages of feeling alcohol’s effects, no matter how mild—your liver is already at its saturation level for protecting the brain. When we talk about “moderation” with alcohol, we miss this crucial point.
Alcohol is toxic in any amount, so the liver’s job is to soak up every last drop it can. When your liver is being poisoned by alcohol, it can’t release glucose anymore. Even if someone has a large storage bin of glucose, eventually the liver becomes paralyzed by the alcohol in this vitally important function—because the liver’s job of releasing glucose is halted. Its job instead becomes soaking up alcohol.
At the same time, alcohol pushes aside any glucose that is in the bloodstream, so alcohol becomes the brain’s number one choice for fuel, because alcohol seems like glucose, even though in the end, it isn’t. Alcohol is a byproduct of glucose. It’s a byproduct of sugar. It’s a ghost of what sugar was. So the brain becomes a victim of the ghost effect of alcohol. As more alcohol enters the brain, a person exhibits more drunken (that is, inebriated) behavior. When someone gets to the point of drunken behavior—slurred speech, inability to function normally—that means the brain is already starting to starve to death. And here’s a key understanding: most of the symptoms we associate with drunkenness and alcohol poisoning aren’t only from alcohol itself. Most are symptoms of the brain starting to die.
The more alcohol you consume in an evening or day, the less glucose the brain absorbs and then fuels itself with. If we think of the optimum as 100 percent of glucose entering the brain and keeping it alive, then drinking alcohol brings that glucose percentage down to 5 to 10 percent, depending on how inebriated someone is. It’s like taking a fish out of water, watching it gasp on the beach as it takes in oxygen instead of water, then putting it back in the water to revive it, and then taking it back out and repeating. The fish will stay alive, although it pays the price for being thrust into survival mode. This is what happens when someone drinks alcohol regularly. Just enough glucose is getting to the brain to keep the person alive, yet it’s so little that the person loses the ability to function. You become a walking, talking example of a dying brain.
Alcohol dominates over glucose getting to the brain because not only is the liver intoxicated and paralyzed and cannot release enough glucose to get to the brain—but the brain chooses alcohol over glucose. This is not because the brain needs alcohol, or because alcohol is good for the brain. Again, it’s because the brain is being tricked into thinking the alcohol is the most accessible, viable form of glucose.
THE INTOXICATION ELEMENT
The effect of alcohol on the brain isn’t solely from this trick sugar effect, where the brain becomes starved of valuable glucose. Alcohol is indeed a poison, and as a poison it does have an effect that can be intoxicating and debilitating. Yet the slurred speech when someone is on their third drink is because the brain is starting to starve from glucose and therefore losing the ability to function.
When inebriation becomes extreme and drastic and someone drinks so much that they collapse, conk out, and fall asleep, that drunken sleep is a game of Russian roulette. Because if the brain doesn’t get any glucose at all due to the intensity of intoxication, the brain can actually starve and that person can die in their sleep. Or due to alcohol poisoning (one aspect of inebriation that’s not about lack of glucose), they may need to vomit. As the brain is dying from a lack of glucose, the nerves are not functioning optimally. The vagus nerves can become paralyzed as the brain is losing deeper glucose reserves, meaning that as someone is vomiting in their sleep, it’s easier to choke and die.
Eating enough glucose-rich foods and keeping your fats low is important before a drinking night so you have ample storage bins of glucose freshly available. This is why when someone says, “I’m drinking on an empty stomach. I haven’t eaten today,” you’ll see them get buzzed faster, showing those first effects of drinking early. We think this buzz, this tipsiness, is because the brain becomes intoxicated with alcohol. Really an alcohol buzz is a brain starving of glucose. The liver is starting to mop up the alcohol, so it’s not releasing glucose anymore, and because someone didn’t eat, they also don’t have freshly available glucose in the bloodstream. For someone who did eat that day, it takes longer for the alcohol to affect them as they start drinking because they at least have that fresh glucose for the brain.
It takes a larger volume of alcohol for intoxication to come into it—that is, for the poison aspect of alcohol to play a role in someone’s symptoms. Even then, drunkenness is part intoxication by alcohol and part brain starving of glucose.
If drunkenness were only about intoxication of alcohol, the symptoms would be limited: someone would be nauseous, vomiting, feel sick in the head, feel dizzy, yet while greatly sick, they would still be coherent. It’s the starvation of the brain happening at the same time that leads to loss of motor skill function, slurred speech and other difficulties speaking, not understanding what someone is saying, and at the same time, saying things you don’t know you’re saying. As the brain is getting very little glucose, on the edge of staying alive, certain parts of the brain start to shut down.
MISGUIDED HANGOVER CURES
A hangover, like drunkenness itself, is part from starvation of glucose, part from intoxication of alcohol. The worst approach for recovery is to drink again the next day. Even though that’s the advice given to many people who drink, it’s the worst option. It doesn’t work. It doesn’t shake a hangover— because once again, you’re starving the brain of glucose.
The reason people tend to gorge on food the day after drinking is because their brain is asking for glucose. Depending on how much alcohol someone consumed, the brain narrowly missed starving to death from lack of glucose, so now the organ sends messaging throughout the body that it needs large amounts of glucose desperately and immediately. At the same time, someone could still be nauseous from the alcohol, feeling sick to the stomach and like they can’t really hold down food yet. That queasy part of a hangover is from the toxic nature of alcohol.
Many people don’t drink to the point of nausea and vomiting. They are still looking to scarf down food to sober up. The same night they drank, they’re looking for a diner or drive-through at two or five o’clock in the morning, ordering a whole stack of pancakes with maple syrup, toast, eggs, bacon, waffles, hash browns, burgers, fries, tacos, or burritos. Or they’re turning to ample food the next day. The common phrase is “sopping up the alcohol.” It even happens when someone is actively drinking, out at a party or a bar when a companion says, “You’ve got to eat something to sop up that alcohol.” What no one realizes is that eating food after drinking alcohol is actually about getting glucose to the brain so it can come out of its starving state and start to function again.
Because we misunderstand what’s going on in the brain and body, we’re still not getting what our brain really needs—because we’re adding fat. Mistakenly, we reach for a combination of carbohydrates plus fat, and that fat inhibits the carbohydrates’ glucose from getting to the brain. For example, frying hash browns in oil, butter, and grease inhibits the potato’s glucose from easily getting to the brain. Now we’re getting insulin resistance, and our body has to fight to divide the sugar and fat so the sugar can get to much- needed places in the brain and other parts of the body. It’s not like we’re trying to recover from a drunken stupor by consuming fruit such as bananas or papayas or mineral salts from sources such as spinach, other leafy greens, or celery juice. Instead we go for a plate of eggs, which is fat. Or we go for toast with avocado, or oatmeal with nut butter—and avocado and nut butter are both fats getting in the way of the glucose from the carbohydrates of the toast or oatmeal. Across the board, pizza (again, sugar plus fat) is often the most popular go-to after a night or even day of drinking.
ADRENALINE SURGE OF SURVIVAL
Why is starving the brain of glucose addictive—that is, why is alcohol addictive? Because there’s an unexpected adrenaline high that comes with the brain losing its fuel source to stay alive. The more alcohol that gets to a brain, in turn starving the brain of glucose, the more adrenaline (epinephrine) is released. This adrenaline can affect each person differently. It can determine if someone is going to be an angry drunk, or if someone is going to be sitting down on the ground crying or outright bawling when they’re drunk.
When we say alcohol is talking for someone, what we’re really witnessing is adrenaline being used by a brain starving to death. Any time we’re in danger on any level, our adrenals send out an adrenaline blend in hopes of changing the chemistry of our bloodstream to help in any way possible. Adrenaline becomes a backup fuel when the brain has no fuel. And remember, adrenaline in itself is an addiction. The more inebriated we get and the more our brain starves of fuel, the more adrenaline is released.
This adrenaline surge often affects people based on what experiences they’ve had in their life. We each feel different emotions when adrenaline is released. Life experiences and wounds tend to peek their heads up when someone drinks. That’s one reason why each person has a different emotional experience when they drink. Some people call alcohol relaxing, some say it gives them a migraine, some say it makes them sad and depressed, some say it makes them happy, some say it gives them strength, energy, and courage. It’s all about how someone’s reacting to the adrenaline surge. Some people get pumped up when they start to drink—excited, screaming, and yelling with their first round of beers, whether cheering at a sports event or simply celebrating that happy hour moment.
They call it “happy hour” for a reason. The sensation that occurs when you’re knocking back that first drink is this adrenaline surge of survival in the face of brain starvation. If we understood this, what we’d be shouting instead of “Cheers!” is “My brain is about to start starving to death! My adrenal glands are going to release a tremendous amount of epinephrine to keep my brain alive! At the same time, I’m going to feel the brain effects of intoxication from alcohol’s poison! All in one, it’s going to give me a great night!” Except it wouldn’t feel like such a great night after all.
要了解更多相关信息,请见以下六本书详细关于现代各项慢性疾病的解说:

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第三本书 关于甲状腺

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第四本 -肝脏救援,肝脏问题是所有疾病的起源

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第五本 关于西芹汁是如何能够疗愈两百多种病症的。

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第六本书关于各种净化疗愈法

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第七本为第一本书的增订版(目前尚无中文版)

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第8和第9本书是2022年10月即将出版《拯救大脑》的英文版

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一本慢性病+难治疾病的食疗全书!看了本书,你会用全新的眼光看待世界与自己的健康!安东尼.威廉从小拥有疗愈人的天赋,至今已成功帮助数万名被误诊,或是因为病怎么治都治不好而受苦的人恢复健康。四岁的某一天,他在吃晚餐时走向奶奶,将手放在奶奶的胸膛,开口说:“肺.癌。奶奶有肺癌。”当时他连“肺癌”两个字都说不清楚,遑论明白意思,奶奶也好端端的,所以家人为此震惊无比。但事后奶奶去照X光,证实果真罹患肺癌。之后,无论遇到什么人,他都能清楚知道对方的身心健康状况,以及需要做些什么来改善。对自己的这项天赋异能,他从好奇、排斥、骄矜、逃避,到完全臣服,进而将此能力奉献给因病所苦之人。现在,他将二十五年来为许多人找回健康的过程中所得知的大量疗愈资讯公诸于世。

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