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·Adult hypoglycemic encephalopathy or hypoglycemic braininjury is caused by an imbalance between supply and use of glucose by cerebralcells, leading to brain injury. The clinical manifestation is characterized byseizures, a depressed level of consciousness, and even coma in patients withdiabetes (commonly in those patients undergoing insulin replacement therapy)·成人低血糖性脑病或低血糖性脑损伤是由脑细胞供血和使用葡萄糖之间的不平衡引起的，导致脑损伤。临床表现的特征是在糖尿病患者（通常在接受胰岛素替代疗法的患者）中发作，意识水平低下甚至昏迷。
·Hypoglycemic encephalopathy has a predilectionfor posterior and deep regions.The most common imaging findings aresymmetric hyperintensities on T2-weighted and FLAIR images and strong restricted diffusion affecting thegyri in the parieto-occipital andtemporal regions on diffusion-weightedimages. The basal ganglia can beinvolved, and this involvement may point to poor outcomes. Another suggestive characteristic finding is the sparing of the thalami, whitematter, and cerebellum (Fig 14). Earliersigns are better depicted on T1-weightedimages as sulcal effacement owing to gyral swelling and can be visualized on CT images ashypoattenuation。·低血糖脑病好发于后部和深部。最常见的影像表现是T2加权像和FLAIR像上对称的高信号，弥散加权像上影响顶枕区和颞区的强限制性扩散。基底节区可能受累，而且这种受累可能表明预后不佳。另一个有提示意义的特征性发现是丘脑、白质和小脑的保留(图14)。早期体征在T1加权图像上较好地显示为脑回肿胀所致的脑沟消退，在CT图像上可显示为低密度。
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